Paulina Carmona-Mora - Publications

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Monocyte, neutrophil, and whole blood transcriptome dynamics following ischemic stroke - BMC Medicine

Early peripheral blood gene expression associated with good and poor 90-day ischemic stroke outcomes - Journal of Neuroinflammation

Distinct peripheral blood monocyte and neutrophil transcriptional programs following intracerebral hemorrhage and different etiologies of ischemic stroke - PubMed

Mitigating losses: how scientific organisations can help address the impact of the COVID-19 pandemic on early-career researchers - Humanities and Social Sciences Communications

Diverse Molecular Mechanisms Contribute to Differential Expression of Human Duplicated Genes

Call to Action: Supporting Latin American Early Career Researchers on the Quest for Sustainable Development in the Region

Creating New Opportunities for Genomics in Central Asia

Integrative Modeling and Novel Technologies in Human Genomics

The nuclear localization pattern and interaction partners of GTF2IRD1 demonstrate a role in chromatin regulation - Human Genetics

RAI1 Transcription Factor Activity Is Impaired in Mutants Associated with Smith-Magenis Syndrome

Functional and cellular characterization of human Retinoic Acid Induced 1 (RAI1) mutations associated with Smith-Magenis Syndrome - BMC Molecular Biology

Retinoic Acid Induced 1, RAI1: A Dosage Sensitive Gene Related to Neurobehavioral Alterations Including Autistic Behavior

Genetic variation contributes to gene expression response in ischemic stroke: an eQTL study

Gene expression changes implicate specific peripheral immune responses to Deep and Lobar Intracerebral Hemorrhages in humans

Syntaxins 6 and 8 facilitate tau into secretory pathways

RNA-Seq analysis of Gtf2ird1 knockout epidermal tissue provides potential insights into molecular mechanisms underpinning Williams-Beuren syndrome - BMC Genomics

Correct developmental expression level of Rai1 in forebrain neurons is required for control of body weight, activity levels and learning and memory

Detection of classical 17p11.2 deletions, an atypical deletion and RAI1 alterations in patients with features suggestive of Smith–Magenis syndrome - European Journal of Human Genetics

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Podocyte-Specific Overexpression of Wild Type or Mutant Trpc6 in Mice Is Sufficient to Cause Glomerular Disease

Mouse Models of Genomic Syndromes as Tools for Understanding the Basis of Complex Traits: An Example with the Smith-Magenis and the Potocki-Lupski Syndromes

Abnormal social behaviors and altered gene expression rates in a mouse model for Potocki-Lupski syndrome

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